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Publications

Molecular Mechanism of Modulation of the TMEM16A Channel by Anthracene-9-Carboxylic Acid: Implications for Channel Gating

Dinsdale R. et al, (2020), BIOPHYSICAL JOURNAL, 118, 325A - 325A

Control of cerebral blood flow by capillary pericytes in Alzheimer's disease

Korte N. et al, (2019), ACTA PHYSIOLOGICA, 227

The structural basis of lipid scrambling and inactivation in the endoplasmic reticulum scramblase TMEM16K.

Bushell SR. et al, (2019), Nat Commun, 10

The Ca2+-gated Cl- channel TMEM16A is a crucial mediator of pericyte contraction in the CNS microvasculature

Korte N. et al, (2019), GLIA, 67, E649 - E649

Inhibition of TMEM16A by Docosahexaenoic Acid Plays a Crucial Role in Blood Vessel Relaxation

Acheson KE. and Tammaro P., (2019), BIOPHYSICAL JOURNAL, 116, 172A - 172A

Defining the ionic mechanisms of optogenetic control of vascular tone by channelrhodopsin-2.

Rorsman NJG. et al, (2018), Br J Pharmacol, 175, 2028 - 2045

Contrasting effects of phosphatidylinositol 4,5-bisphosphate on cloned TMEM16A and TMEM16B channels.

Ta CM. et al, (2017), Br J Pharmacol, 174, 2984 - 2999

Mechanism of allosteric activation of TMEM16A/ANO1 channels by a commonly used chloride channel blocker.

Ta CM. et al, (2016), Br J Pharmacol, 173, 511 - 528

Corrigendum to "Disease-causing mutations associated with four bestrophinopathies exhibit disparate effects on the localization, but not the oligomerization, of Bestrophin-1" [Exp. Eye Res. 121 (2014) 74-85] DOI: 10.1016/j.exer.2014.02.006

Johnson AA. et al, (2014), Experimental Eye Research, 127

Disease-causing mutations associated with four bestrophinopathies exhibit disparate effects on the localization, but not the oligomerization, of Bestrophin-1.

Johnson AA. et al, (2014), Exp Eye Res, 121, 74 - 85

Coronary spasm and acute myocardial infarction due to a mutation (V734I) in the nucleotide binding domain 1 of ABCC9.

Smith KJ. et al, (2013), Int J Cardiol, 168, 3506 - 3513

Putative pore-loops of TMEM16/anoctamin channels affect channel density in cell membranes.

Adomaviciene A. et al, (2013), J Physiol, 591, 3487 - 3505

Autosomal dominant hypercalciuria in a mouse model due to a mutation of the epithelial calcium channel, TRPV5.

Loh NY. et al, (2013), PLoS One, 8

Parametrisation of the free energy of ATP binding to wild-type and mutant Kir6.2 potassium channels.

Moran O. et al, (2013), Biophys Chem, 171, 76 - 83

Molecular Modelling and Experimental Analysis of the ATP Sensitivity of Cloned Smooth Muscle KATP Channels

Chadburn A. et al, (2012), BIOPHYSICAL JOURNAL, 102, 539A - 539A

TMEM16A/anoctamin 1 protein mediates calcium-activated chloride currents in pulmonary arterial smooth muscle cells.

Manoury B. et al, (2010), J Physiol, 588, 2305 - 2314

Vascular K ATP channels: dephosphorylation and deactivation.

Tammaro P., (2009), Br J Pharmacol, 157, 551 - 553

A cytosolic factor that inhibits KATP channels expressed in Xenopus oocytes by impairing Mg-nucleotide activation by SUR1.

Tammaro P. and Ashcroft FM., (2009), J Physiol, 587, 1649 - 1656

Journal club. A physiologist notes the similarities between animal and plant electricity.

Tammaro P., (2009), Nature, 458

Variable phenotypic spectrum of diabetes mellitus in a family carrying a novel KCNJ11 gene mutation.

D'Amato E. et al, (2008), Diabet Med, 25, 651 - 656

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