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Publications

Endolysosomal calcium release and cardiac physiology.

Terrar DA., (2022), Cell Calcium, 104

Inhibition of adenylyl cyclase 1 by ST034307 inhibits IP3-evoked changes in sino-atrial node beat rate.

Bose SJ. et al, (2022), Front Pharmacol, 13

Emerging evidence for cAMP-Ca2+ cross talk in heart atrial nanodomains where IP3-evoked Ca2+ release stimulates adenylyl cyclases

BURTON R-A. and Derek T., (2021), Sage Open

Calcium Signaling in the Heart.

Terrar DA., (2020), Advances in experimental medicine and biology, 1131, 395 - 443

Synthesis of the Ca2+-mobilizing messengers NAADP and cADPR by intracellular CD38 enzyme in the mouse heart: Role in β-adrenoceptor signaling.

Lin WK. et al, (2017), J Biol Chem, 292, 13243 - 13257

Two-pore Channels (TPC2s) and Nicotinic Acid Adenine Dinucleotide Phosphate (NAADP) at Lysosomal-Sarcoplasmic Reticular Junctions Contribute to Acute and Chronic β-Adrenoceptor Signaling in the Heart.

Capel RA. et al, (2015), J Biol Chem, 290, 30087 - 30098

Contributions of cardiac "funny" (f) channels and sarcoplasmic reticulum Ca2+ in regulating beating rate of mouse and guinea pig sinoatrial node.

Nazarov IB. et al, (2015), Physiol Rep, 3

Living cardiac tissue slices: an organotypic pseudo two-dimensional model for cardiac biophysics research.

Wang K. et al, (2014), Prog Biophys Mol Biol, 115, 314 - 327

Cardiac electrophysiological imaging systems scalable for high-throughput drug testing.

Lee P. et al, (2012), Pflugers Arch, 464, 645 - 656

β-Adrenergic receptor signaling increases NAADP and cADPR levels in the heart.

Lewis AM. et al, (2012), Biochem Biophys Res Commun, 427, 326 - 329

ß-Adrenergic receptor signaling increases NAADP and cADPR levels in the heart

Lewis AM. et al, (2012), Biochemical and Biophysical Research Communications, 427, 326 - 329

Ca(2+)-stimulated adenylyl cyclases regulate the L-type Ca(2+) current in guinea-pig atrial myocytes.

Collins TP. and Terrar DA., (2012), J Physiol, 590, 1881 - 1893

NAADP influences excitation-contraction coupling by releasing calcium from lysosomes in atrial myocytes.

Collins TP. et al, (2011), Cell Calcium, 50, 449 - 458

Comparison of guinea-pig ventricular myocytes and dog Purkinje fibres for in vitro assessment of drug-induced delayed repolarization.

Terrar DA. et al, (2007), J Pharmacol Toxicol Methods, 56, 171 - 185

Dual effects of cyclic ADP-ribose on sarcoplasmic reticulum Ca2+ release and storage in cardiac myocytes isolated from guinea-pig and rat ventricle.

Macgregor AT. et al, (2007), Cell Calcium, 41, 537 - 546

NAADP controls cross-talk between distinct Ca2+ stores in the heart.

Macgregor A. et al, (2007), J Biol Chem, 282, 15302 - 15311

Regulation of L-type calcium channel and delayed rectifier potassium channel activity by p21-activated kinase-1 in guinea pig sinoatrial node pacemaker cells.

Ke Y. et al, (2007), Circ Res, 100, 1317 - 1327

Actions of a membrane permeant analogue of NAADP on contractions and calcium sparks in cardiac ventricular myocytes.

Rakovic S. et al, (2007), BIOPHYSICAL JOURNAL, 594A - 594A

NAADP: a novel Ca2+ mobilizing messenger during beta-adrenoceptor stimulation in guinea-pig ventricular myocytes

Yamasaki M. et al, (2007), BIOPHYSICAL JOURNAL, 365A - 365A

Possible novel mechanism for the positive inotropic action of alpha-1 adrenoceptor agonists in guinea-pig isolated atrial myocytes.

Collins TP. et al, (2007), BIOPHYSICAL JOURNAL, 433A - 433A

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